Jennifer Lake's Blog

August 4, 2009

Bacteriophage II

Filed under: DNA - RNA,Medicine — jenniferlake @ 6:55 pm
Tags: , , , , ,
Bacteriophage II
Statements from the website above:
…”If the devil were to work overtime in a plot to confound scientists in their quest for a causative agent to a serious disease of unknown origin, it is doubtful whether he could have come up with anything better than the phage …It is little wonder then why scientists could easily mistake bacterial elements for viruses…”
…”by the 1950s the word “virus” had become so mouldable a concept that one could speak of virus workers without the existence of any concensus whatsoever of what viruses were. Extremely supportive of this mouldability among virologists was Max Delbruck’s subtitle for Virus, 1950, a conference held at [Cal Tech] called “Proceedings of a conference on the similarities and dissimilarities between viruses attacking animals, plants, and bacteria.”
…”Indeed, in the 1930s and 1940s the concept of ‘filterable virus’ was subjected to such criticism that its very foundations were threatened…”
…”Statements coming from pioneer virologist Andre Lwoff in 1957 such as “viruses should be considered as viruses because viruses are viruses” were totally unacceptable. Also under the gun was Thomas M. Rivers, the father of American Virology…Rivers was wrong…some bacteria could pass through filters that some of the larger viruses could not.”
In the 1918 Pandemic…”the death rate was twice as high among men…the current epidemic wasn’t just influenza, thought [William] Welch. Nor did he believe those who speculated a ‘filterable virus’ which passed through a porcelain or clay filter. These reports had not been reproducible…..this ‘Influenza’s’ clinical symptoms truly shocked medical officers across the country… Patients could spike to 105 or 106 F with accompanying delirium..bled from the nose and swollen lungs exuded bloody froth, causing pathologists to say, over and over again, that this must be some kind of new disease… Nevertheless, virologists saw a great opportunity provided by the 1918 pandemic and by hook or by crook would prove it to be viral. Richard Shope began the first salvo working on ‘swine influenza’… But beginning his investigations in earnest, Shope became perplexed. Not a virus but a bacteria kept cropping up…and it resembled the Pfeiffer’s bacillus, or Hemophilus Influenzae, more than anything else. The problem was he couldn’t infect most of his subjects with the bacteria…”
“There is an old adage which rings particularly true for even state of the art science. It reads “You get what you look for”. It is also a trap that no scientist should fall into. Specifically, if you are looking for a viral cause for the lethal Pandemic of 1918 and ignoring any evidence of bacteria.. you will not only ignore evidence of bacterial disease in specimens but you will use agents such as phenol which can kill every bacteria that crawls. You will be sure that the ‘virus’ passes a filter.. to once and for all nail down your argument. But does that mean in reality what you have seen and labeled a virus is in certainty a virus? Not at all. The fact that tuberculosis killed an estimated 1 billion people by itself between 1850 and 1950, the approximate midpoint of which was the Pandemic, is immaterial…”
“Bacteria reproduce asexually so there is no variation of genes in a colony, and no way for them to exchange genetic material. Phages, the viruses which live inside bacteria such as the mycobacteria called tuberculosis, can allow exchange of this genetic material, as well as force natural selection. The way bacteriophages, or ‘phages’, allow gene diversity is after they insert their own genetic material into their bacterial prey’s genome, they in essence hijack the manufacturing capacity of the bacteria for their own purposes, reproducing new viruses in the bacteria….. When these viruses infect other bacteria, usually of the same class, such as from bird tuberculosis to human tuberculosis inside the pig, they add the other bacteria’s genes to the new bacteria. [this was also done ‘in vitro’ in laboratories by flu researchers –JL]. This creates genetic diversity or ‘mutations’. Such genetic mutations can create far more virulent bacteria than either of the parent bacteria. This is what is theorized happened in 1918 Haskell County, Kansas, where genetic elements of human and avian tuberculosis combined inside hogs just prior to the Pandemic of 1918″.
“Dr. Andrew Noymer and Michel Garenne, UCBerkely demographers, reported in 2000 convincing statistics showing that undetected tuberculosis may have been the real killer in the 1918 flu epidemic… It is theorized that the lethal pig epidemic that began in Kansas just prior to the first human outbreaks was a disease of avian and human tuberculosis genetically combined through mycobacteriophage interchange… The obvious need for further investigation is presently imminent and pressing.”
[end excerpts]
I posit that one would have to ignore many of the other possibilitites besides ‘phage’ and ‘flu genes’ brought about by toxic conditions in 1918. Clearly, ‘filtrate’ was an unreliable method of producing ‘purified’ phage and virus although such experimentation was well advanced by the onset of WWI. X-rays, radium treatment, toxic medicants, experimental vaccines, blood products derived from animals, malnutrition, crowding and stress, etc. all contributors to the many factors of flu. Human enteroviruses carried into the bloodstream and organs are just as able to mutate and infect the lungs and the possibilties of intentional or accidental bioweapons cannot be ruled out.

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